高度近視的影響因素是什么?

　　遺傳因素基在高度近視中起到了很大的作用。然而，近視不是一種簡單的遺傳性疾病，它可以受到各種基因的影響。它還受到外部環境因素的影響。

　　因此，如果高度近視的父母有計劃保護他們的視力，就很難想象高度近視;即使你沒有，你的下一代也可能患有高度近視。

　　高度遠視不要劇烈運動

　　如果你是高度近視的，你應該避免劇烈的運動，特別是那些有直接影響眼睛和頭部或有明顯的腦震蕩。例如，潛水或蹦極，拳擊，跳高，甚至在遊樂園玩一些暴力搖滾遊戲。

　　新鏡片設有用作矯正 視力及散光的「中心光學區」，並於圍繞中心光學區伸延至鏡片周邊加入小區域離焦度數，令眼內形成「近視離焦」，令小童的眼軸生長會趨向變短，有效抑制配戴者近視加深。

　　但我們現在要說的是，如果你想打籃球，足球，羽毛球和其他運動，這基本上是可能的，但我們必須小心，不要傷害我們的眼睛。

　　高度遠視,更隨便馬虎發生眼底病變

　　高度遠視以至可能引起視網膜脫離。然而，我們咨詢了眼科醫生，沒有發現高度近視患者不獻血或分娩的證據或原因，但高度近視患者更有可能患視網膜病變。

　　這些病變也會導致失明!目前，如果沒有良好的治療方法，視力可能會變得非常差，戴眼鏡也行不通。

　　高度近視怎樣可以恢複?

　　注意眼、腎等眼病，即中醫對肝髒的認識;中藥治療羊肝、石斛發光、明目地黃丸等，均按高元治療。然而，這些近視治療並不理想。根本原因是由長期眼肌疲勞引起的近視。問題不在於深部器官，而在於經絡的層次。認識到這一點，近視很容易解決。

　　醒目的視覺康複中心致力於發展全國青少年的視覺健康。通過調節患者的身體功能，消除肌肉疲勞，促進全身氣血自律性的恢複。普通的按摩或藥物很難減輕眼部肌肉的負擔。在醒目視力康複中心療養近視患者有很好的康複效果，從而避免高度近視的發展和摘下眼鏡，看得更清楚!

　　高度近視、眼部曾因外力損傷、家族遺傳性基因等人群是這類眼部疾病的高發人群，定期是預防眼部疾病的最好方式。

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　　支持系統不斷升級，業務環境不斷優化。

　　商務部最近公布的數據顯示，7月份我國實際使用外資4967.1億元，比去年同期增長2.3%。外國投資的吸引力穩步增長，結構繼續優化。實現小的生長是困難的。面對複雜、多變的外部環境，如何實現吸引外國資本的穩定增長?如何繼續提高穩定的外資質量和增量?記者在廣東和福建采訪。

　　保護知識產權等外商投資企業的合法權益

　　“這不是為了保護弱點。公平地說，埃洛·史密斯的經曆使我真正感到，地方政府重視保護產權，同時也重視維護外國企業的合法權益。”澳史密斯(中國)熱水器公司副總裁朱洪蘭說 。

　　多年前，Ao Smith的產品被廣東中山的一家公司侵犯，Ao Smith選擇在另一個地方起訴。起初，他們認為侵權案件或多或少會遇到曲折。無非，朱紅蘭說他們更關心。 Ao Smith在南京提起的另一起侵權案被轉移到廣東中山。成功超越了想象、責任和獎勵。與以往國外訴訟中遇到的困難相比，中山地方訴訟更為方便。

　　廣東在維護外商投資企業合法權益方面的積極探索是顯而易見的。近年來，廣東省高等法院公布的十起加強財產權司法保護的案件中，一件曆時20多年的老案件引起了廣泛關注。

　　1997年，揭陽市公安局查封了飛利浦船運公司的WUSA號貨輪，涉嫌走私，並沒收了所攜帶的貨物。飛利浦公司支付了100000美元的保安費。當時，幾乎所有人都認為這是無可指責的。直到2014年1月，揭陽市公安局決定取消WUSA號貨輪的涉嫌走私案件。

　　廣東省高等法院認為，公安機關要嚴格依法行使職權，平等保護一切經濟實體的合法權益。該案件是一個國家賠償案件，長期以來一直被撤銷，給受害企業造成了巨大的經濟損失。揭陽公安局撤銷了對涉嫌走私烏薩的調查，對非法扣押貨船造成的損害承擔賠償責任，有權依法得到國家賠償。

　　最初，廣東省高等國民法院抉擇，揭陽市公安局補償菲利普的包管款項和本錢，船舶設備和設備損壞修理費和差旅費等損失129萬元，並支付了66萬元的集裝箱賠償金。這不是為了保護短缺，不是掩蓋醜陋，善待舊案，公開，反映了廣東維護中國外資企業合法權益，營造良好商業環境的決心。

　　我們將恒生指數成份股比重按其每日股價表現順序排列，方便投資者掌握並準備輪證部署。此頁面亦提供每一隻恒數成分股之比重及當日的成交額及市值供投資者參考。

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中國網財經9月18日訊據商務部網站消息，上周(9月10日至16日)全國食用農產品市場價格比前一周上漲1.6%，生產資料市場價格上漲0.5%。

食用農產品市場：30種蔬菜平均批發價格為每公斤4.51元，比前一周上漲4.4%，其中菜花、青椒、苦瓜價格漲幅居前，比前一周分別上漲16.8%、12.3%和12%。肉類批發價格繼續上漲，其中豬肉、牛肉、羊肉價格分別為每公斤21.03元、53.46元和50.45元，比前一周分別上漲2.5%、0.4%和1%。禽產品批發價格總體上漲，其中雞蛋、白條雞、白條鴨價格分別為每公斤10.26元、16.37元和17.54元，比前一周分別上漲0.9%、0.7%和1%。糧食批發價格小幅上漲，其中面粉、大米價格比前一周分別上漲0.3%和0.2%。水產品批發價格延續跌勢，其中小帶魚、小黃魚、大黃魚等海水魚價格跌幅居前，比前一周分別下跌3.3%、1.2%和0.6%。食用油批發價格略有下降，其中花生油價格比前一周下降0.3%；豆油、菜籽油價格均與前一周基本持平。

生產資料市場：基礎化學原料價格比前一周上漲1.6%，其中硫酸、甲醇、燒堿價格分別上漲2.6%、1.6%和1.1%，純堿價格保持平穩。橡膠價格上漲0.5%，其中天然橡膠價格上漲1%，合成橡膠價格下降0.2%。化肥價格上漲0.5%，其中尿素、氯化鉀、三元複合肥、磷酸二銨價格分別上漲0.6%、0.4%、0.4%和0.3%。鋼材價格上漲0.4%，其中無縫鋼管、高速線材、螺紋鋼、普通圓鋼價格分別為每噸5144元、4476元、4381元和4447元，分別上漲0.7%、0.5%、0.5%和0.4%。有色金屬價格上漲0.3%，其中銅、鎳、鉛價格分別上漲1.1%、1%和0.4%，鋁、鋅、錫價格分別下降0.7%、0.2%和0.1%。煤炭價格上漲0.1%，其中動力煤價格為每噸598元，上漲0.2%，無煙煤價格為每噸862元，下降0.1%，煉焦煤價格為每噸749元，與前一周持平。

原文地址：http://field.10jqka.com.cn/20180918/c607177047.shtml

Drinking Water Is Necessary To Switch Your Fat Burning Enzymes On
Fat breakdown depends on water. It doesn’t happen efficiently without plenty of it. Looking at your body fat as a whole, something has to happen to it for it to shrink so you can get slimmer. It needs to be broken down so it can be used by your body.

An enzyme called lipase is required to break down fat. If you imagine your fat as a brick wall, lipase is a chisel that removes the bricks one by one. The bricks are now free to travel to be used around the body and burned up for energy.

Lipase needs water in a good supply to work well. This is how it works:

Fat + Water + Lipase = Fatty Acids

Fatty acids are the small building blocks of fat. They are small enough to be released into your circulation and will be picked up by cells around your body needing energy. They are released easier when you are well hydrated. Drinking water to lose weight allows the enzymes to break down fat efficiently.

The findings, published by the journal Circulation, helps explain why some patients experience a heart attack or the need for coronary revascularization to open blocked heart arteries while taking statins. It suggests that drugs targeting the genetic variations could lower the heart risk in these patients.

The study demonstrates the power of genome-wide association studies and longitudinal electronic health records (EHRs) to find links between genetic variation and disease, said the paper's first author, Wei-Qi Wei, MD, phD, assistant professor of Biomedical Informatics in the Vanderbilt University School of Medicine .

Some of the patients were followed for heart disease for up to a decade after starting on their statin drug. The study found that the effect of the genetic variations or variants was independent of how much their cholesterol improved while taking statins.

"people with these genetic variants were at a higher risk for heart disease, even considering those who have ideal cholesterol levels on their statin," said Joshua Denny, MD, MS, Vice president of personalized Medicine at Vanderbilt University Medical Center (VUMC) and the paper's corresponding author.

The researchers searched four sites in the Electronic Medical Records and Genomics (eMERGE) network, a nationwide consortium of experts, biorepositories and electronic medical record systems supported by the National Institutes of Health (NIH), including BioVU, VUMC's DNA databank.

They found 3,099 people who had experienced a heart attack or the need for revascularization while on statins, and compared them to 7,681 "control" patients on statins who did not experience heart events.

From this comparison, the researchers were able to identify seven genetic variations, called single nucleotide polymorphisms or SNps, in the LpA locus of genes that were associated with these heart events in patients receiving statin treatment.

The LpA gene encodes apolipoprotein (a), a fatty protein that binds to low-density lipoprotein (LDL), the form of blood cholesterol that is the target of statin drugs. High levels of bound LDL, called Lp(a) for short, is well known to be an independent risk factor for heart disease.

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One of the SNps was highly associated with an increased risk of heart events. When the researchers examined the full EHRs of 11,566 individuals who carried the SNp for more than 1,000 physical conditions, they found significantly higher rates of coronary heart disease and heart attack but not of other diseases.

The approach, called a phenome-wide association study, was pioneered by Denny and his colleagues at Vanderbilt.

"The study highlights the need to consider targeting Lp(a) levels as an important independent factor to reduce cardiovascular risk in patients on statin therapy," Wei concluded.

Efforts to reduce Lp(a) levels using existing or new drugs could reduce heart events in the proportion of patients on statins who carry LpA variations, he added, although clinical trials would be needed to detect potential side effects and confirm the safety of any such treatment.

Thirty-three researchers from 15 academic medical centers and research institutes in the United States and Yokohama, Japan, contributed to the study, including Vanderbilt faculty members Dan Roden, MD, C. Michael Stein, MBChB, Nancy Cox, phD, Todd Edwards, phD, Qiping Feng, phD, and Jonathan Mosley, MD, phD.

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